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Arachnoiditis / Spinal Injuries Manifestaion of other symptems.

#1 User is offline   Tomcat 

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Posted 02 November 2005 - 10:58 AM

GREETINGS,
Back/ Spinal injuries…
Arachnoiditis can occur from either Injury, or injections, or injected substances…
AND SURGERY...
This condition is not an easy one to diagnose…
As many with Spinal injuries, will know, it is not just the pain, but other symptoms, will manifest… eg. Irritable Bowel Syndrome. I.B.S…. Lung problems… Indigestion etc. Sciatica and other organ disfunction..
In a lot of cases it is the medication that is the main cause.
And sometimes the treatment / medication for an”apparent condition” is not necessary.

All this can be very stressful and frustrating…


But in a lot of cases, after these symptoms are “investigated”, it will come down to the cause is damage to spine and nerves, as the cause…
(But rarely put down to the condition of Arachnoiditis…)
A point worth discussing with your GP.



There is much info here… and worth a read thru…

http://www.accforum....hp?showforum=15

I have had various complaints / symptoms “investigated”… only to come to the conclusion, with GP in agreement, that the cause originates from the spine and the injury(s)… Knowing the why and how, is a “relief in itself”…
The use of herbal remedies, and vitamin and mineral supplements, goes a long way to counter the negative effects on the body and mind.

Down load the spine zip… from “shock wave”… pin points the locations in the spine that effect things elsewhere.

TC.
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#2 User is offline   batman 

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Posted 02 November 2005 - 05:19 PM

People with spinal injuries or back pain please note there is an Arachnoiditis Sufferers Action and Monitoring Society (ASAMS) in New Zealand who provide information and support for all chronic back pain and associated conditions. To join or for further information contact Steve Emslie, 39 Jackson St, Wanganui, ph 06 3477573, fax 06 3488470, website http://www.arachnoiditis.info, email [email protected] They provide a 2 monthly booklet on pain, coping with pain etc etc, which I have found to be very good reading and quite informative. There is a small joining fee.
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#3 User is offline   gaffa09 

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Posted 04 November 2005 - 09:32 AM

Yes I know about ASAM

I was the founder of ASAM forming the group back in the early 1990s after exposing myodil, Arachnoiditis and other drugs through the dominion news paper then going on to TV 60 minutes program, also on radio through the whole of NZ,
I also have travelled to gather infomation and have been in contact with C Burton

So I can tell you what i think of ASAM,

We want help but from Government level and medical provides

Have a good day
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#4 User is offline   gaffa09 

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Posted 17 February 2006 - 09:43 AM

Does Myodil cause brain damage ,

Myodil , a diagnostic drug injected into spines use for x-rays .

But if it travels to far in your spine and it lands up in your brain , Then someone has made a blunder ,
Myodil is supposed to be aspirated , { REMOVED }
Back in the mid 60s warning where given about the dangers .

The world experts say and have recorded the adverse reactions to this drug which was taken off the market 1987 , It has a shelf life of 3 years so by 1990 it should not have been used ,
My understanding is that it now comes under the health Dept as from 1987 ,
Before this time the health dept had no due astriction over this drug.

In My thousands of pages that I have on the dangers of myodil it is well recorded on the side effects ,

It is now recognised by the health dept on the effects ,

Yet ACC do not accept that it causes problems when it is lodged in the brain.
ACC ignore all medical notes over a long period of time that spell it out.
GP know nothing about Arachnoiditis ,
Yet this disease Arachnoiditis has been found in humans far back in time as the cave man days . .
Arachnoiditis can be caused by just a smack on the back, or whip lash , from a car accident .,
There are different types of arachnoiditis as recorded .
But man made chemically induced arachnoiditis , which Glaxo knew about back in 1947 of the effects .
The pain , the effects that it has on your life and the people around you is unreal ,

Does it cause brain damage WELL my medical notes say it does ,,
My friends and family that see me often know that something is wrong , my GP knows something is wrong ,
Yet before my accident I never had these problems ,
A list of medical problems I have are already posted for all to read.
Also what causes these wicked adverse reaction are also listed. Not only Myodil but many other drugs ,
Please search the site and read THIS MAY EFFECT YOU TO
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#5 User is offline   gaffa09 

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Posted 28 February 2006 - 06:06 PM

Acute Brachial Plexus Neuritis: An Uncommon Cause of Shoulder Pain
JIMMY D. MILLER, M.D., STEPHANIE PRUITT, R.N., and THOMAS J. MCDONALD, M.D.,
North Mississippi Neurosurgical Services, P.A., Tupelo, Mississippi
Patients with acute brachial plexus neuritis are often misdiagnosed as having cervical radiculopathy. Acute brachial plexus neuritis is an uncommon disorder characterized by severe shoulder and upper arm pain followed by marked upper arm weakness. The temporal profile of pain preceding weakness is important in establishing a prompt diagnosis and differentiating acute brachial plexus neuritis from cervical radiculopathy. Magnetic resonance imaging of the shoulder and upper arm musculature may reveal denervation within days, allowing prompt diagnosis. Electromyography, conducted three to four weeks after the onset of symptoms, can localize the lesion and help confirm the diagnosis. Treatment includes analgesics and physical therapy, with resolution of symptoms usually occurring in three to four months. Patients with cervical radiculopathy present with simultaneous pain and neurologic deficits that fit a nerve root pattern. This differentiation is important to avoid unnecessary surgery for cervical spondylotic changes in a patient with a plexitis. (Am Fam Physician 2000;62:2067-72.)

Acute brachial plexus neuritis is an uncommon disorder of unknown etiology that is easily confused with other neck and upper extremity abnormalities, such as cervical spondylosis and cervical radiculopathy.1-3 Patients with acute brachial plexus neuritis present with a characteristic pattern of acute or subacute onset of pain followed by profound weakness of the upper arm and amyotrophic changes affecting the shoulder girdle and upper extremity.1,2,4 In 1943, Spillane5 was probably the first to recognize acute brachial plexus neuritis as a distinct clinical entity. In 1948, Parsonage and Turner6 described 136 cases of this condition and, in view of the doubts of pathology and etiology at the time, gave it the name "neuralgic amyotrophy."

Illustrative Case

A 66-year-old man presented with the complaint of severe, left-sided neck pain that radiated into the left shoulder, without associated numbness or tingling. Onset of the pain was one week earlier. Several weeks before he presented for examination, he had received an influenza vaccination. On initial physical examination, he exhibited normal strength, sensation and reflexes of the upper extremities. He subsequently developed weakness in the deltoid, infraspinatus and supraspinatus muscles. His sensory examination was within normal limits.


Acute brachial plexus neuritis mimics cervical radiculopathy in several respects, but the treatment is significantly different.


Cervical spine radiographs revealed cervical spondylosis. Magnetic resonance imaging (MRI) did not reveal nerve root compression on the left; however, it did reveal a spondylotic defect on the right at the C4-5 level and a bony ridge at the C5-6 level, causing foraminal narrowing that was greater on the right than on the left. The patient underwent evaluation with a myelogram, and a computed tomographic (CT) myelogram revealed osteophytes bilaterally at the C4-5 level and lesser spondylotic changes at the C3-4 level.

Cerebrospinal fluid studies revealed 1,045 red blood cells per mm3 (1,045 X 106 per L) with 3 nucleated cells per mm3 (3 X 106 per L), a glucose of 53 mg per dL (2.94 mmol per L), and a slightly elevated protein of 60 mg per dL (0.60 g per L).

Approximately three to four weeks after the onset of symptoms, the patient underwent an electromyogram and nerve conduction studies. The results were consistent with a diagnosis of brachial plexus neuritis with severe subacute denervation in the supraspinatus, infraspinatus and deltoid muscles. In particular, moderately severe spontaneous fibrillations and positive waves were evident in the deltoid, supraspinatus, infraspinatus and low cervical paraspinous muscles, and moderate, chronic repetitive discharges were noted in the levator scapulae muscles. With voluntary muscle activation, motor unit action potentials were of normal amplitude, duration and contour. Distal median and ulnar motor and sensory and radial sensory nerve conduction studies were within normal limits, as were proximal median and ulnar motor nerve conduction studies. A diagnosis of acute brachial plexus neuritis was made.

The patient was treated with a methylprednisolone dosepak (Medrol), hydrocodone and cyclobenzaprine (Flexeril). He underwent physical therapy for three weeks, and his condition slowly improved; however, he still experienced some mild difficulty with shoulder abduction for several months.

Discussion
Pain in the cervical and shoulder area is common and may reflect a multitude of conditions. However, when a patient develops neurologic deficits, the list of differential diagnoses becomes shorter. Cervical radiculopathy is the most common condition. Patients with cervical radiculopathy present with cervical pain and neurologic deficits resulting from a herniated nucleus pulposus or an osteophyte. Acute brachial plexus neuritis mimics cervical radiculopathy in several aspects, but the treatments are significantly different.

Etiology and Epidemiology

Acute brachial plexus neuritis has been recognized as a distinct plexus disorder since the 1940s. Multiple names have been ascribed to it, including "brachial plexus neuropathy," "local neuritis of the shoulder girdle," "acute brachial plexitis," "acute shoulder neuritis," "paralytic neuritis" and "Parsonage-Turner syndrome."

A viral etiology has been proposed, while other studies have emphasized that various infections precede the onset of acute brachial plexus neuritis in as many as 25 percent of cases.3,7,8 Up to 15 percent of cases have been reported to occur following vaccinations, including hepatitis B vaccination.6,9 Our patient received an influenza vaccination before the onset of brachial plexus neuritis. Some evidence suggests that acute brachial plexus neuritis may be an immunologic disease.10

Most cases of acute brachial plexus neuritis occur between 20 and 60 years of age; however, cases have been reported in all age groups. A male predominance is reported, with a male-to-female ratio ranging from 2:1 to 11.5:1.3,4,11 The annual incidence has been estimated as 1.64 cases per 100,000 persons, but this figure is probably low because many cases may be misdiagnosed, or the symptoms are mild and clinically unrecognized.12 It is not uncommon for patients to present with bilateral acute brachial plexus with only one side being symptomatic.13

Clinical Presentation


The hallmark clinical presentation of acute brachial plexus neuritis is severe, acute burning shoulder pain with no apparent cause.


The hallmark clinical presentation of patients with acute brachial plexus neuritis is severe, acute, burning pain in the shoulder and upper arm with no apparent cause. On occasion, it may awaken the patient from sleep.1,2,4 In the majority of patients, the pain subsides over the ensuing days to weeks, resulting in a subsequent weakness in the upper arm--at times to the point of muscle flaccidity.2,3,11 This temporal profile of initial arm and shoulder pain followed by muscle weakness as the pain subsides is an important characteristic of acute brachial plexus neuritis.

The usual abnormality evident on physical examination is one of a brachial plexus lesion, as indicated by involvement of two or more nerves14 (Figure 1). Weakness commonly occurs in the supraspinatus, infraspinatus, deltoid and/or the biceps muscles usually involving the upper plexus. However, isolated or single nerve involvement has been clinically reported.15 The course of the neuritis is usually one of gradual improvement and recovery of muscle strength in three to four months.2 Some patients, however, experience several years of muscle weakness or a slight permanent weakness. In general, a longer duration of pain will result in a longer delay in recovering strength.13


FIGURE 1. Acute brachial plexus neuritis usually involves the upper plexus, which supplies the shoulder and upper arm muscles. Weakness is frequently found in the rhomboideus major and minor muscles (dorsoscapular nerve), supraspinatus and infraspinatus muscles (suprascapular nerve), deltoid muscle (axillary nerve) and biceps muscle (musculocutaneous nerve).

Differential Diagnosis

Patients with cervical radiculopathy present with pain beginning in the neck area and radiating down the arm for variable distances. The pain may occur after a documented trauma but, not uncommonly, patients may awake in the morning with the pain and no obvious preceding etiology. The pain is associated with partial weakness in the muscles supplied by the involved nerve root and sensory loss in the appropriate dermatome (Tables 1 and 2).


TABLE 1
Comparison of Acute Brachial Plexus Neuritis and Cervical Radiculopathy
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Condition
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History
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Examination
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Tests and results
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Treatment
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Acute brachial plexus neuritis Intense, burning pain begins in shoulder and upper arm. Pain is unaltered by neck or arm movements.

Pain is spontaneous, often with no apparent cause.

Gradual decrease in pain followed by marked weakness of upper arm. Neurologic deficits indicate that more than one nerve is involved (i.e., lesion in the plexus). Electromyography and nerve conduction studies obtained three to four weeks after symptom onset reveal abnormalities consistent with a brachial plexus lesion.

MRI scan of the clinically weak muscles may reveal high signal intensity on T2 images; these changes may appear within days following onset of symptoms. Analgesics as needed for pain

Physical therapy to maintain strength and mobility

If deltoid muscle is profoundly weak, recommend a sling to avoid subluxation of humerus.

Encourage patient that condition usually, but slowly, improves.
Cervical radiculopathy Pain begins in neck and radiates down the arm for variable distances.

Pain is aggravated by neck movements.

Pain may begin spontaneously following physical exertion or trauma, but may have no apparent cause.

Pain and muscle weakness occur simultaneously. Weakness and numbness in the distribution of a single nerve root (contemporaneous with the neck and arm pain) Cervical spine radiograph may reveal interspace narrowing and osteophytes.

MRI scan or myelogram followed by computed tomographic scan may reveal osteophyte or herniated disc pulposus consistent with clinical findings. Analgesics as needed for pain

Steroid therapy may help decrease nerve root irritation.

Muscle relaxants for muscle spasms

Physical therapy

Massage and cervical traction

Anterior and posterior surgical procedures to decompress involved nerve roots

--------------------------------------------------------------------------------

MRI = magnetic resonance imaging.



Unlike acute brachial plexus neuritis, the pain, weakness and sensory loss associated with cervical radiculopathy tend to occur simultaneously. While acute brachial plexus neuritis involves multiple nerves of the brachial plexus, a radiculopathy by definition is restricted to one nerve root.


TABLE 2
Cervical Radiculopathy Patterns
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Nerve root involvement
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Muscle involvement
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Sensory area
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Reflex
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C5 Deltoid (shoulder abduction) Cap of shoulder None
C6 Biceps (elbow flexion) Thumb and index finger Biceps
C7 Triceps (elbow extension) Middle finger Triceps



Diagnostic Evaluation

MRI
In many ways, the use of MRI has improved neuroradiologic evaluation of diverse conditions, including those that involve the peripheral nervous system. In patients with acute brachial plexus neuritis, MRI of the clinically weak muscles may reveal high signal intensity of the affected muscles on the T2 study.2 These changes may appear within days following the onset of symptoms and persist for months. A delayed MRI scan may also reveal muscle atrophy.2 An MRI scan of the plexus and muscles of the shoulder girdle or upper arm is seldom required to establish a diagnosis, but it may be useful if an early, specific diagnosis would be beneficial.

With a typical presentation and an examination suggesting nerve root involvement, the diagnosis of cervical radiculopathy may be confirmed by a myelogram followed immediately by a cervical CT scan. More commonly, however, patients undergo an MRI scan of the cervical spine.

Electromyography
Electromyographic testing in patients with acute brachial plexus neuritis yields variable data, depending on the severity of neural damage and the timing of the examination. It localizes the lesion to the brachial plexus (usually involving the upper aspect of the plexus), and physicians often use results of this test along with a patient history and physical examination to establish the diagnosis. In most cases, three weeks following the onset of paresis, a needle electrode examination will reveal fibrillation potentials and positive waves suggestive of muscle denervation.1 Nerve conduction studies of the medial and ulnar nerves are generally within normal limits. Patients will often recover strength in the denervated muscles approximately three to four months following the initial presentation. This period is characterized by giant polyphasic potentials.1

Treatment


Differentiating acute brachial plexus neuritis from other diagnoses is important so that surgical treatment is not performed for small osteophytes that may be observed on magnetic resonance imaging but are not causing neurologic deficits.


Treatment of patients with acute brachial plexus neuritis includes analgesics, often narcotics (e.g., hydrocodone), which may be required for several weeks, physical therapy for three to eight weeks to help maintain strength and mobility, and encouragement that the condition will slowly improve in the vast majority of patients. The profound weakness in the shoulder muscles may require the use of a sling. Corticosteroids, although frequently used, are not of proven benefit.

Final Comment

Differentiation of acute brachial plexus neuritis from cervical radiculopathy may be problematic in some patients, but it usually is apparent by conducting a careful patient history and performing a neurologic examination. This will separate the single nerve root finding in cervical radiculopathy from the "multiple nerve" findings of a brachial plexus lesion.

Findings on cervical MRI may be helpful if the study is normal or if there is evidence of appropriate nerve root compression, indicating cervical radiculopathy. Difficulty occurs when there are changes of spondylosis at multiple levels because one may be lulled into thinking these radiographic abnormalities account for the clinical deficits. Electromyography and nerve conduction studies are useful, especially when combined with a patient history and physical examination findings, but characteristic changes of a plexus abnormality may not be apparent for three weeks following the onset of symptoms. Differentiating acute brachial plexus neuritis from other diagnoses is important so that surgical treatment is not performed for small osteophytes that may be present on MRI, but are not causing the patient's neurologic deficits.


--------------------------------------------------------------------------------

The Authors

JIMMY D. MILLER, M.D.,
is in private practice at North Mississippi Neurosurgical Services, P.A., Tupelo. Dr. Miller received his medical degree from the University of Mississippi, Jackson. He completed a residency in general surgery and a residency in neurosurgery at Mississippi Medical Center, Jackson.

STEPHANIE PRUITT, R.N.,
is in private practice at North Mississippi Neurosurgical Services, P.A., Tupelo. She received her nursing degree from Northeast Mississippi Community College, Booneville.

THOMAS J. MCDONALD, M.D.,
is in private practice at North Mississippi Neurosurgical Services, P.A., Tupelo. Dr. McDonald received his medical degree from the University of Mississippi, Jackson, and completed a residency in neurosurgery at the University of Mississippi Medical Center, Jackson.

Address correspondence to Jimmy D. Miller, M.D., North Mississippi Neurosurgical Services, 812 Garfield St., Tupelo, MS 38801. Reprints are not available from the authors.

REFERENCES

Aymond JK, Goldner JL, Hardaker WT. Neuralgic amyotrophy. Orthop Rev 1989;18:1275-9.
Helms CA, Martinez S, Speer KP. Acute brachial neuritis (Parsonage-Turner syndrome): MR imaging appearance--report of three cases. Radiology 1998;207:255-9.
Misamore GW, Lehman DE. Parsonage-Turner syndrome (acute brachial neuritis). J Bone Joint Surg Am 1996;78:1405-8.
Turner J. Acute brachial radiculitis. BMJ 1944;2: 592-4.
Spillane JD. Localized neuritis of the shoulder girdle. A report of 46 patients in the MEF. Lancet 1943;2:532-5.
Parsonage M, Turner J. Neuralgic amyotrophy: the shoulder-girdle syndrome. Lancet 1948;1:973-8.
Tsairis P, Dyck PJ, Mulder DW. Natural history of brachial plexus neuropathy. Report on 99 patients. Arch Neurol 1972;27:109-17.
Pellas F, Olivares JP, Zandotti C, Delarque A. Neuralgic amyotrophy after parvovirus B19 infection [Letter]. Lancet 1993;342:503-4.
Reutens DC, Dunne JW, Leather H. Neuralgic amyotrophy following recombinant DNA hepatitis B vaccination [Letter]. Muscle Nerve 1990;13:461.
Sierra A, Prat J, Bas J, Romeu A, Montero J, Matos JA, et al. Blood lymphocytes are sensitized to branchial plexus nerves in patients with neuralgic amyotrophy. Acta Neurol Scand 1991;83:183-6.
Turner JW, Parsonage MJ. Neuralgic amyotrophy (paralytic brachial plexus neuritis) with special reference to prognosis. Lancet 1957;2:209-12.
Beghi E, Kurland LT, Mulder DW, Nicolosi A. Brachial plexus neuropathy in the population of Rochester, Minnesota, 1970-1981. Ann Neurol 1985;18:320-3.
Dillin L, Hoaglund FT, Scheck M. Brachial neuritis. J Bone Joint Surg Am 1985;67:878-80.
Miller JD. Clinical evaluation of the brachial plexus: a simplified approach. J Miss State Med Assoc 1987;28:141-3.
Wong L, Dellon AL. Brachial neuritis presenting as anterior interosseous nerve compression--implications for diagnosis and treatment: a case report. J Hand Surg [Am] 1997;22:536-9.
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#6 User is offline   gaffa09 

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Posted 27 March 2006 - 01:16 PM

Still checking out the site ACC
Why don;t you do something about it

The Facts are here.! :angry:

http://www.accforum....hp?showforum=15
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#7 User is offline   MadMac 

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Posted 27 March 2006 - 06:07 PM

:wub: Hey gaffa09 ... it takes a while for some people to learn how to say the ... biiiiiiiiiiiiiiiiiiiiiiiiiiig ... medicial words in the picture...

:wacko: Let alone understand what they mean ... could be part of the process to educate some people so they have some form of knowledge of what they are talking about ...

:D Facts in front of your face ... common mate thats tooooooo easy ...

;)
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#8 User is offline   MadMac 

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Posted 18 April 2008 - 06:05 PM

:wub:

XXX

;)
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#9 User is offline   practioner123 

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Posted 20 January 2013 - 03:39 AM

Interesting that I find a discussion about arachnoiditis. I didn't even think people knew what it was, so interesting to find it on this site. Dr Burton has a great website about this very real condition.

I do a technique that I think helps relieve it. Arachnoiditis refers to the arachnoid layer, surrounding the spinal cord. It represents an inflammation of the spinal cord fibers, themselves, and can be visualized on MRI. The technique I use was based on the research of a neurosurgeon from Sweden, Dr Alf Brieg. After learning this technique (only taught in the States, as for as I know), my first patient, who could hardly walk for 14 years, had her first pain free day in 14 years, following 6 months of regular treatment. Another, who came in with an MRI, showing obviously inflamed cauda equina, had his first day free of low back pain in years after 10 treatments (was on holiday). I could go on and on.

What's interesting about the technique is that it involves function of the entire spinal cord, not just in the low back, but the neck and skull, because the entirety of the brain and spinal cord is covered in a continuous ligament called the "dura mater." It's as if your brain and spinal cord is encased in a flexible steel sheet, and it all moves as one unit. One of the fascinating discoveries of that neurosurgeon, working on fresh cadavers, cut in half, was that when the neck loses its curve, it pulls the spinal cord all the way down tot he tailbone, and the cord itself loses up to 7 cm of slack. That means, if you pull the collar of your shirt, does it not pull all the way down to the bottom, where you have it tucked into your jeans? Your spinal cord functions the same way. You pull the neck, by simply pushing your head forward, you also pull your low back region of the spinal cord, pulling on the nerves, that get stretched and no longer function (there's your bowel issues, pain down the leg, etc).

The sad thing is that while I got accolades from one claims officer at ACC, and said she would send what I was doing up the flag pole at ACC, for further recommendation, it seems the rest of ACC doesn't seem to give a s-it. Nothing ever came of the officer's accolade, after having helped someone with a serious head injury. It's sad, because a lot of people who are injured could get better with this technique. Some people take longer than others, but I'd have to say, the only ones who didn't get better are usually the ones who quit, and/or don't follow the dietary recommendations (cut everything made from seeds, especially breads, cereals, and margarine, and increase your animal fat intake).

This is the problem with a system that is designed to fail, like ACC. It's a symptom of the condition of our so-called 'healthcare" model, as well. There is no real intention on fixing things, I feel, but rather "management" of pain, and injured parties. While I can appreciate ACC's position that there are plenty of "dole bludgers" out there who would like nothing more than to sit back and collect ACC for the rest of their lives, there are plenty that would love nothing else than to carry on with their lives, the same as it was before the injury. Instead it appears there is no search for an increase in quality in the health of the individual, or genuine research into what works and what doesn't, and I find it sad. People suffer. When they get to my office they are often broke or destitute, like the one above, who after 14 years of not being able to work, and seeing over 50 specialists, including psychiatrists, and having been cut from ACC, had to borrow the money to pay for my treatment. It makes you angry.

How many other people could be helped? What is ACC doing to research what works and what doesn't? I believe their official stance is to push people off the system once they believe they have done all that known medical science can do for you. It becomes your problem, not theirs, and selfish humans as we are, that bonus is due. Gotta meet that quota. It is why I came here. I am looking for any information about bonuses for ACC staff. Anything you have. Do they use "hay points?" KPI? Or did they do away with that, and replaced it with something else, that you must specify in your Official Information Act request? Does the investigations department receive any incentives? It seems that ACC finds it easier to accuse a person of fraud than to actually do their duty and help the person get better. If they don't think they can, then instead of admitting defeat, or exploring different options (like what I'm doing), they make it your problem. It's such short term thinking. "The purpose of government is to prevent injustice, except for the injustice caused by the government itself." Ibn Khalduhn, father of economics and sociology.

So many people would not need surgery if they had this technique, I believe. A lot of the "pain management" programs would become un-necessary, not to mention surgery. I've had patients who had been through those programs, and were no better when they were done. Yet, they were told "it's all in your head," which I can understand be made out of frustration, when you don't know what to do, but if a person has tension on their spinal cord, pain is a very fair conclusion. Drugs will not fix a mechanical tension in the spinal cord brought about by physical injury. And no, this is not just another "manipulation." I hate that word. This technique involves stretching of the dura in a way that you cannot do yourself. It's rather intense, and can be uncomfortable. However, this must be measured against having a life of pain. These stretches are painless for a person with no issues, and eventually you reach that stage, depending on how messed up you are, how many injuries you've accumulated over your lifetime.

After what I've seen, I would never have back surgery- ever, if I could help it, because I think most of them can be fixed without surgery, and surgery is irreversible, whereas with this, the worst that can happen is it will delay surgery. Also, people in pain are under the mistaken belief that surgery will give them the miracle they seek, and "do something!" Sadly it doesn't work out that way. One surgery often (if not usually) leads to another, later. There is a structural/bio-mechanical reason why this is. When you fixate a lower joint with surgery (usually at L5-S1), then your L4-L5 disk must carry the new burden for what was once a moveable joint at L5-S1, so it too blows out. Surgery at L5-S1 begets surgery at L4-L5 about 5 years later.

I will say this, I have never met a back pain patient who didn't have a neck injury first. Fall off the horse, or get in an auto accident today, and 15-20 years later you can't get away from your low-back pain. This is typical, and there is a structural reason why. All of this effects the flow of the precious fluid that nourishes your spinal cord, and if it is impeded, the cord can't receive nourishment or flush out toxin. This is "cerebra-spinal fluid." If the dura mater is stretched, it's like stretching a hose. It becomes thinner. The fluid flows in the space between the spinal cord and the dura. If the space becomes thinner, then the fluid can't flow, and you get arachnoiditis, essentially. This is a simplistic explanation, but a major factor. There is also tension on the filum terminal, affecting the cauda equina, as well. All of this is mechanical tension.

The tension works both ways. A person with tension in their low back will also have tension in their neck, because the cord is continuous. I've had tremendous results with migraine headaches using this technique, and we work on the low back as par tof the treatment. It's intense, and I've had several people quit. It's not easy for some people, especially those that have really old injuries, fibromyalgia, etc, however, the ones that stay with it usually do get better, in my experience. The ones that don't get better is usually because they quit, or they really don't want to get better (they've come to identify with their injury, or use it as a means to curry sympathy from a spouse, etc). The ones intent on getting better usually do. Some take longer than others, well, because they are more messed up than they think. Surprisingly, some of the most messed-up people I've treated have been health practitioners, and I've treated a few. You would think that they would have the healthiest spines, but sadly they don't. I myself had a bit of pain, having been through 2 auto accidents in succession, and I'm happy to say I am pain free now.

As an aside, I treated that guy who climbed to the top of Christchurch tower to protest his ACC treatment. He's much better now. Talked to him a year after his treatment, and he says he's still doing well. His problem was missed by about a dozen practitioners of various types. He did receive injections, and ACC refused his surgery. He didn't need surgery or injections (injections are a waste of time IMO). Have to catch up with how it's going with his home. Destroyed in quake.

I came to this forum while doing a search regarding incentives for ACC employees. I did not post because I thought that ACC controlled the board, and I did not hear back from someone after I requested some information, so I thought maybe the communication got ambushed by an ACC employee. Now I understand that this board is handled by members of the public, so I can speak more freely.

I'm in a bit of a different situation, as a treatment provider. I have my own problems with ACC. If you have any information that proves ACC staff, especially the investigation department, receives bonuses for doing certain things, it would be most appreciated. I get the impression that they are trying some of the same tactics now, against successful clinics, as they employ against "expensive" patients. The sad thing is, that a lot of these "expensive" patients, I think can be fixed, and a successful clinic should be left alone to do its job, and ACC would, in fact, do well to send all of their trouble patients to us because we can probably get a lot of them back on their feet- and "rehabilitate" them for real, instead of sending them to some medical whore who says they can drive a tractor. Our average patient has "been everywhere else, tried everything." Have heard it all before. One of our biggest challenges is the medications. My gosh, some people have a lot, all fixing nothing, making the patient dependent. It slows down progress greatly, plus withdrawl issues. A little challenge there, especially the cholesterol meds.
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